Stina Rudolfsson

Publications

Author

Title

Year sorteringsordning

Fulltext

Halin Bergström, Sofia
Rudolfsson, Stina H.
Bergh, Anders

Rat Prostate Tumor Cells Progress in the Bone Microenvironment to a Highly Aggressive Phenotype
Neoplasia, 18(3): 152-161

2016

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Hammarsten, Peter
Winther, Johanna
Rudolfsson, Stina H; et al.

ErbB2 Receptor Immunoreactivity in Prostate Cancer: Relationship to the Androgen Receptor, Disease Severity at Diagnosis and Disease Outcome
PLoS ONE, 9(9): e105063-

2014

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Rudolfsson, Stina

Att hämma tumörtillväxt

2013

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Jernberg, Emma
Thysell, Elin
Bovinder Ylitalo, Erik; et al.

Characterization of prostate cancer bone metastases according to expression levels of steroidogenic enzymes and androgen receptor splice variants
PLoS ONE, 8(11): e77407-

2013

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Johansson, Anna
Rudolfsson, Stina Häggström
Kilter, Sigrid; et al.

Targeting castration-induced tumour hypoxia enhances the acute effects of castration therapy in a rat prostate cancer model
BJU International, 107(11): 1818-1824

2011

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Hammarsten, Peter
Karalija, Amar
Josefsson, Andreas; et al.

Low levels of phosphorylated epidermal growth factor receptor in nonmalignant and malignant prostate tissue predict favorable outcome in prostate cancer patients.
Clinical Cancer Research, 16(4): 1245-1255

2010

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Johansson, Anna
Rudolfsson, Stina
Hammarsten, Peter; et al.

Mast cells are novel independent prognostic markers in prostate cancer and represent a target for therapy
American Journal of Pathology, 177(2): 1031-1041

2010

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Halin, Sofia
Häggström Rudolfsson, Stina
Doll, Jennifer A; et al.

Pigment epithelium-derived factor stimulates tumor macrophage recruitment and is downregulated by the prostate tumor microenvironment
Neoplasia, 12(4): 336-345

2010

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Halin, Sofia
Häggström Rudolfsson, Stina
Van Rooijen, Nico; et al.

Extratumoral macrophages promote tumor and vascular growth in an orthotopic rat prostate tumor model.
Neoplasia, 11(2): 177-186

2009

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Rudolfsson, Stina Häggström
Bergh, Anders

Hypoxia drives prostate tumour progression and impairs the effectiveness of therapy, but can also promote cell death and serve as a therapeutic target.
Expert opinion on therapeutic targets, 13(2): 219-225

2009

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