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Umeå University
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Åsa Stenberg

Contact

E-mail
asa.stenberg@umu.se
Phone

Works at

Affiliation
Laboratory technician at Department of Medical Biosciences
Location
By 6M, Sjukhusområdet Umeå universitet, 901 85 Umeå
Opposing roles by KRAS and BRAF mutation on immune cell infiltration in colorectal cancer: possible implications for immunotherapy
British Journal of Cancer
Edin, Sofia; Gylling, Björn; Li, Xingru; et al.
Studies of SIRPα-mediated regulation of neutrophil functions
Umeå University medical dissertations, 1630
Stenberg, Åsa
Signal regulatory protein alpha is present in several neutrophil granule populations and is rapidly mobilized to the cell surface to negatively fine-tune neutrophil accumulation in inflammation
Journal of Innate Immunity, Vol. 6, (4) : 553-560
Stenberg, Åsa; Karlsson, Anna; Feuk-Lagerstedt, Elisabeth; et al.
Lack of CD47 impairs bone cell differentiation and results in an osteopenic phenotype in vivo due to impaired signal regulatory protein α (SIRPα) signaling
Journal of Biological Chemistry, Vol. 288, (41) : 29333-29344
Koskinen, Cecilia; Persson, Emelie; Baldock, Paul; et al.
Neutrophil apoptosis is associated with loss of signal regulatory protein alpha (SIRP alpha) from the cell surface
Journal of Leukocyte Biology, Vol. 93, (3) : 403-412
Stenberg, Åsa; Sehlin, Janove; Oldenborg, Per-Arne
Osteoclast formation is strongly reduced both in vivo and in vitro in the absence of CD47/SIRPalpha-interaction
Biochemical and Biophysical Research Communications - BBRC, Vol. 352, (2) : 444-448
Lundberg, Pernilla; Koskinen, Cecilia; Baldock, Paul A; et al.
Deficit of CD47 results in a defect of marginal zone dendritic cells, blunted immune response to particulate antigen and impairment of skin dendritic cell migration.
Journal of Immunology, Vol. 176, (10) : 5772-8
Hagnerud, Sven; Manna, Partha Pratim; Cella, Marina; et al.
Signal regulatory protein alpha (SIRPα) negatively regulates neutrophil migration by stimulating β2 integrin-mediated adhesion
Stenberg, Åsa; Sehlin, Janove; Matozaki, Takashi; et al.
View publications in DiVA

Research groups

Group member
Richard Palmqvist Lab